Just imagine all those tiny, almost invisible hairs all over your scalp.
What is clear is that identical thing happens in aging men and women.
The result. Anagen stage of hair growth gets shorter, and the resting stage gets longer. Hair follicles get smaller. Keep reading.a lot of us, when we think of hair loss, think about aging men. Nearly all men eventually get that receding ‘Mshaped’ hairline and thinning hair on the top of the head, in addition known as male pattern baldness. One aftereffects of Propecia can be loss of libido. Alopcia areata isn’t associated with a more serious condition known as cicatricial alopecia, in which the immunity attacks the stem cells in the bulge of the folicle. On top of that, it keeps the male sex hormone testosterone from forming its DHT ‘byproduct’.
And now here’s a question. Why?
The other currently approved drug for hair loss is Propecia.
It works only for men. For the most part there’re other hair loss experts like me working in the field who are racing to get this treatment out as a product. However, aging women have a similar problem. Similarly out of fashion are flap type procedures, where a flap of hair from a hair bearing area is partially removed, swung around, and attached to a frontal area.
Why is this pattern of hair loss only in the front and on top? That’s where hormone sensitive follicles live. Those new treatments aren’t nearly ready for prime time, like the cure for cancer. Today, Americans spend $ 800 million on hair restoration surgery. Actually, most of us know that there are good reasons this kind of technology will move forward. And so it’s, Therefore in case this all sounds futuristic. On top of that, hair follicles live just below the top layer of the skin. Seriously. They don’t divide like normal cells, in which both halves become new cells that split and developing. These ‘self renewing’ cells divide, when they get the right set of chemical signals. Now regarding the aforementioned fact… Only one the follicle half stem cell does that. Therefore, that’s where follicle stem cells live. For example, the ‘IGF1’ ain’t nearly as effective, without this. So it is a vital part of the puzzle. So, already they’ve made a major breakthrough. Essentially, that’s what Cotsarelis’s lab is working on. Eventually, the Holy Grail of hair loss treatment is getting shutdown follicles to regenerate.